医学前沿学术报告第177期—Endogenous mechanisms of neuroprotection for the treatment of stroke

题 目：Endogenous mechanisms of neuroprotection  for the treatment of stroke

主讲人：Roger Simon

Director, Translational Programs in Stroke
Professor, Medicine (Neurology) and Neurobiology

Morehouse School of Medicine

主持人：颜光美教授

            中山大学 副校长

            中山医学院 药理教研室教授

时   间：2014年4月30日（星期三）上午10:00时

地   点：中山大学北校区永生楼4楼讲学厅

主讲人简介：

Dr. Roger Simon earned his M.D. degree from Cornell University in 1971 and subsequently trained in Neurology at the University of California San Francisco. He remained in San Francisco for twenty years to become Professor and Vice Chairman of Neurology. In 1994 he moved to the University of Pittsburgh, where he was Professor and Chairman of Neurology and in 1999 he became the first Chair and Director of R.S. Dow Neurobiology Laboratoriesat Legacy Health Research in Portland, Oregon. He has been at Morehouse School of Medicine in Atlanta as Professor of Medicine and Neurobiology since 2010.His early work in stroke defined glutamate neurotoxicity in ischemia (Simon, Science, 1984; a paper cited > 1000 times). More recent work addressed ischemic tolerance mechanisms in brain. These studies, showed that tolerance could attenuate ischemic injury from stroke and offered the first molecular mechanistic data in tolerance, demonstrating that gene down regulation was the phenotype of the tolerant brain (Stenzel-Poore, Lancet,2003). Recently the lab has used unbiased, quantitative proteomics to define the proteome in ischemic tolerance (Stapels, Science Signaling, 2010).These proteins, members of the polycomb protein group (PcG), are epigenetic regulators of transcription.

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                                                       2014年 4月21日